Case reportIdiopathic Tracheal Stenosis: Successful Outcome With Antigastroesophageal Reflux Disease Therapy
Section snippets
Comment
Idiopathic laryngotracheal stenosis includes lesions that share typical features of location, configuration, clinical evolution, and pathologic findings. This patient had some of these features, as well as female sex, fifth decade of life, and upper tracheal stenosis, with no history of intubation, trauma, infection, or collagen vascular disease (eg, Wegener granulomatosis or relapsing polychondritis). Therefore, a diagnosis of ILTS was made [1]. Because of good results of surgical treatment in
References (6)
- et al.
Idiopathic laryngotracheal stenosis and its management
Ann Thorac Surg
(1993) - et al.
Idiopathic laryngotracheal stenosis: effective definitive treatment with laryngotracheal resection
J Thorac Cardiovasc Surg
(2004) - et al.
Association of laryngopharyngeal reflux disease and subglottic stenosis
Ann Otol Rhinol Laryngol
(2001)
Cited by (18)
Management of laryngopharyngeal reflux in Brazil: a national survey
2022, Brazilian Journal of OtorhinolaryngologyCitation Excerpt :In Asia, otolaryngologists recognize an association between Reinke’s edema and LPR but not with nasal obstruction, chronic and acute otitis media and laryngotracheal stenosis.5 In the literature, clinical and experimental studies suggested that LPR could be associated with laryngotracheal stenosis,8–10 acute otitis media11,12 and Eustachian dysfunction.12 The role of LPR in the development of benign lesions of the vocal folds, especially nodules, Reinke’s edema and polyps, is also highly probable.13
Commentary: The role of gastroesophageal reflux in patients with acquired tracheal stenosis
2019, Journal of Thoracic and Cardiovascular SurgeryImpact of fundoplication for gastroesophageal reflux in the outcome of benign tracheal stenosis
2019, Journal of Thoracic and Cardiovascular SurgeryCitation Excerpt :The incidence of dysphagia was less than 15%, and it was not different between patients with normal and abnormal esophageal manometry findings. Unlike other airway or lung diseases, such as asthma,4,5 pulmonary fibrosis,3 bronchiolitis obliterans,19,20 and airway hyperresponsiveness, the few studies21-23 on the correlation between airway stenosis and GER are focused on idiopathic subglottic airway stenosis where the presence of gastric juice yields to tissue remodeling through the stimulation of the differentiation of fibroblasts into myofibroblasts.24 As far as postintubation tracheal stenosis is concerned, the treatment of reflux and its impact on the outcome of the stenosis have been scarcely reported.
Non-malignant central airway obstruction
2014, Archivos de BronconeumologiaCitation Excerpt :It occurs mainly in women, suggesting that estrogens have an important role in this entity.118–121 Other authors suggest that it may be associated with GERD.122,123 Although evaluation of the flow-volume loop may suggest the diagnosis, multi-slice CT and bronchoscopy (Fig. 1, image 7) are essential for confirmation.124–126
Single-stage subchordal resection and reconstruction of idiopathic laryngotracheal stenosis in a male patient
2012, Journal of Thoracic and Cardiovascular SurgeryCitation Excerpt :However, Dedo and associates2 and others3 failed to find estrogen receptors in the stenotic tissue samples. Gastroesophageal reflux disease has also been considered in the etiology of ILTS,3 with some reports of improvement of ILTS with antireflux therapy.4 Continued rarity of ILTS cases despite continuously increasing gastroesophageal reflux disease and intensive endoscopic surveillance programs, however, do not support the role of reflux in etiology of ILTS.
Endoscopic management of idiopathic tracheal stenosis
2011, Annals of Thoracic SurgeryCitation Excerpt :The etiology of ITS is unknown, but several physiopathologic hypotheses have been proposed. Gastroesophageal reflux (GERD), chronic cough, and an abnormal response to estrogen have been discussed as etiologic factors [2, 18, 19]. Because of cricoid localization of ITS, chronic cough was discussed as inducing local inflammation and ischemia by telescoping of the cricoid cartilage and first tracheal ring [2].